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Emilio Boada-Romero

Researcher at University of Salamanca

Publications -  17
Citations -  7741

Emilio Boada-Romero is an academic researcher from University of Salamanca. The author has contributed to research in topics: Autophagy & ATG16L1. The author has an hindex of 13, co-authored 16 publications receiving 5867 citations. Previous affiliations of Emilio Boada-Romero include St. Jude Children's Research Hospital.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

The clearance of dead cells by efferocytosis.

Emilio Boada-Romero, +3 more
- 06 Apr 2020 - 
TL;DR: The mechanisms of efferocytosis are outlined, from the recognition of dying cells through to phagocytic engulfment and homeostatic resolution, and the pathophysiological consequences that can arise when this process is abrogated are highlighted.
Journal ArticleDOI

LC3-associated endocytosis facilitates β-amyloid clearance and mitigates neurodegeneration in murine Alzheimer’s Disease

Bradlee L. Heckmann, +8 more
- 25 Jul 2019 - 
TL;DR: The data support a protective role for LANDO in microglia in neurodegenerative pathologies resulting from β-amyloid deposition, and identify a novel non-canonical function of several autophagy proteins in the conjugation of LC3 to Rab5+, clathrin+ endosomes containing β-amide.
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LC3-Associated Phagocytosis in Myeloid Cells Promotes Tumor Immune Tolerance.

Larissa D. Cunha, +15 more
- 04 Oct 2018 - 
TL;DR: It is found that the anti-tumor effects of LAP impairment require tumor-infiltrating T cells, dependent upon STING and the type I interferon response, and autophagy proteins in the myeloid cells of the tumor microenvironment contribute to immune suppression of T lymphocytes by effecting LAP.