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Sara Marinelli

Researcher at National Research Council

Publications -  58
Citations -  8417

Sara Marinelli is an academic researcher from National Research Council. The author has contributed to research in topics: Neuropathic pain & Allodynia. The author has an hindex of 26, co-authored 51 publications receiving 6719 citations. Previous affiliations of Sara Marinelli include Novartis & San Antonio River Authority.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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TRPV1 channels are critical brain inflammation detectors and neuropathic pain biomarkers in mice

Maria Cristina Marrone, +16 more
- 10 May 2017 - 
TL;DR: Findings identify brain TRPV1 as potential detector of harmful stimuli and a key player of microglia to neuron communication in microglial cells rather than neurons of the anterior cingulate cortex and other brain areas.
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Denervation-activated STAT3–IL-6 signalling in fibro-adipogenic progenitors promotes myofibres atrophy and fibrosis

Luca Madaro, +16 more
- 26 Jul 2018 - 
TL;DR: It is shown that denervation induces accumulation of IL-6–STAT3-activated fibro-adipogenic progenitors without inflammation or muscle regeneration, leading to muscle atrophy and fibrosis.
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The function neutralizing anti-TrkA antibody MNAC13 reduces inflammatory and neuropathic pain.

Gabriele Ugolini, +4 more
- 20 Feb 2007 - 
TL;DR: It is demonstrated that MNAC13, the only anti-TrkA monoclonal antibody for which function neutralizing properties have been clearly shown both in vitro and in vivo, induces analgesia in both inflammatory and neuropathic pain models, with a surprisingly long-lasting effect in the latter.