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Julio Madrigal-Matute

Researcher at Albert Einstein College of Medicine

Publications -  50
Citations -  9086

Julio Madrigal-Matute is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Autophagy & Inflammation. The author has an hindex of 30, co-authored 46 publications receiving 7226 citations. Previous affiliations of Julio Madrigal-Matute include Complutense University of Madrid & New York University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Effects of Sex, Strain, and Energy Intake on Hallmarks of Aging in Mice

Sarah J. Mitchell, +51 more
- 14 Jun 2016 - 
TL;DR: The authors' data illustrate the complexity of CR in the context of aging, with a clear separation of outcomes related to health and survival, highlighting complexities of translation of CR into human interventions.
Journal ArticleDOI

Regulation of Liver Metabolism by Autophagy

Julio Madrigal-Matute, +1 more
- 01 Feb 2016 - 
TL;DR: 3 mechanisms by which hepatic autophagy monitors and regulates cellular metabolism are reviewed and how these connections might serve as a therapeutic target in common metabolic disorders are discussed.
Journal ArticleDOI

The CD163-expressing macrophages recognize and internalize TWEAK: potential consequences in atherosclerosis.

Juan Antonio Moreno, +8 more
- 01 Nov 2009 - 
TL;DR: The results suggest that TWEAK-CD163 interaction takes place in vivo, probably decreasing TWEak plasma concentration and that CD163-TWEAK plasma ratio is a potential biomarker of clinical and subclinical atherosclerosis.