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Niels Jessen

Researcher at Aarhus University Hospital

Publications -  237
Citations -  15608

Niels Jessen is an academic researcher from Aarhus University Hospital. The author has contributed to research in topics: Skeletal muscle & Insulin resistance. The author has an hindex of 48, co-authored 215 publications receiving 12666 citations. Previous affiliations of Niels Jessen include Steno Diabetes Center & Aarhus University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

High-Dose Resveratrol Supplementation in Obese Men: An Investigator-Initiated, Randomized, Placebo-Controlled Clinical Trial of Substrate Metabolism, Insulin Sensitivity, and Body Composition

Morten Poulsen, +9 more
- 01 Apr 2013 - 
TL;DR: The lack of effect disagrees with persuasive data obtained from rodent models and raises doubt about the justification of resveratrol as a human nutritional supplement in metabolic disorders.
Journal ArticleDOI

Distinct Signals Regulate AS160 Phosphorylation in Response to Insulin, AICAR, and Contraction in Mouse Skeletal Muscle

Henning F. Kramer, +8 more
- 01 Jul 2006 - 
TL;DR: AS160 may be a point of convergence linking insulin, contraction, and AICAR signaling, while Akt and AMPK α2 activities are essential for AS160 phosphorylation by insulin and A ICAR, respectively, neither kinase is indispensable for the entire effects of contraction on AS160osphorylation.
Journal ArticleDOI

Contraction signaling to glucose transport in skeletal muscle

Niels Jessen, +1 more
- 01 Jul 2005 - 
TL;DR: Recent studies in transgenic and knockout animals show that AMP-activated protein kinase is not the sole mediator of the signal to GLUT4 translocation and suggest that there may be redundant signaling pathways leading to contraction-stimulated glucose transport.