scispace - formally typeset
M

Mireia Niso-Santano

Researcher at University of Extremadura

Publications -  59
Citations -  10749

Mireia Niso-Santano is an academic researcher from University of Extremadura. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 27, co-authored 53 publications receiving 8766 citations. Previous affiliations of Mireia Niso-Santano include Carlos III Health Institute & French Institute of Health and Medical Research.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Self-consumption: the interplay of autophagy and apoptosis

Guillermo Mariño, +3 more
- 01 Feb 2014 - 
TL;DR: The dialogue between autophagy and cell death pathways influences the normal clearance of dying cells, as well as immune recognition of dead cell antigens, and the disruption of the relationship between autphagy and apoptosis has important pathophysiological consequences.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

An immunosurveillance mechanism controls cancer cell ploidy

Laura Senovilla, +88 more
- 28 Sep 2012 - 
TL;DR: It is reported that hyperploid cancer cells become immunogenic because of a constitutive endoplasmic reticulum stress response resulting in the aberrant cell surface exposure of calreticulin, which causes recognition of cancer cells in mice by the host immune system.
Journal ArticleDOI

Cytoplasmic STAT3 represses autophagy by inhibiting PKR activity.

Shensi Shen, +35 more
- 14 Dec 2012 - 
TL;DR: An unsuspected mechanism of autophagy control that involves STAT3 and PKR as interacting partners is unraveled, which facilitates autophagic induction.