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Sami Dridi

Researcher at University of Arkansas

Publications -  160
Citations -  10900

Sami Dridi is an academic researcher from University of Arkansas. The author has contributed to research in topics: Broiler & Medicine. The author has an hindex of 31, co-authored 138 publications receiving 8725 citations. Previous affiliations of Sami Dridi include Katholieke Universiteit Leuven & Catholic University of Leuven.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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DICER1 deficit induces Alu RNA toxicity in age-related macular degeneration

Hiroki Kaneko, +43 more
- 17 Mar 2011 - 
TL;DR: Findings reveal a miRNA-independent cell survival function for DICER1 involving retrotransposon transcript degradation, show that Alu RNA can directly cause human pathology, and identify new targets for a major cause of blindness.
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DICER1 Loss and Alu RNA Induce Age-Related Macular Degeneration via the NLRP3 Inflammasome and MyD88

Valeria Tarallo, +30 more
- 11 May 2012 - 
TL;DR: It is shown that DICER1 deficit or Alu RNA exposure activates the NLRP3 inflammasome and triggers TLR-independent MyD88 signaling via IL18 in the RPE and reveals a function of the inflammaome outside the immune system and an immunomodulatory action of mobile elements.
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Alternatively spliced vascular endothelial growth factor receptor-2 is an essential endogenous inhibitor of lymphatic vessel growth

Romulo Albuquerque, +24 more
- 01 Sep 2009 - 
TL;DR: The existence of a splice variant of the gene encoding vascular endothelial growth factor receptor-2 that encodes a secreted form of the protein, designated soluble VegFr-2 (sVegfr-2), that inhibits developmental and reparative lymphangiogenesis by blocking Vegf-c function is reported.