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Paloma Martín-Sanz

Researcher at Spanish National Research Council

Publications -  124
Citations -  11517

Paloma Martín-Sanz is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: Hepatocyte & Proinflammatory cytokine. The author has an hindex of 35, co-authored 116 publications receiving 9589 citations. Previous affiliations of Paloma Martín-Sanz include Carlos III Health Institute & University of Barcelona.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Substrate Fate in Activated Macrophages: A Comparison between Innate, Classic, and Alternative Activation

Juan-Carlos Rodríguez-Prados, +7 more
- 01 Jul 2010 - 
TL;DR: Whether specific metabolic patterns depend on the signaling pathway activated is analyzed, and it is shown that activated macrophages are essentially glycolytic cells, and a clear cutoff between the classic/innate activation and the alternative pathway exists.
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Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD

Águeda González-Rodríguez, +15 more
- 01 Apr 2014 - 
TL;DR: The results demonstrated that the autophagic flux is impaired in the liver from both NAFLD patients and murine models ofNAFLD, as well as in lipid-overloaded human hepatocytes, and it could be due to elevated ER stress leading to apoptosis.
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Inhibition of IκB Kinase and IκB Phosphorylation by 15-Deoxy-Δ12,14-Prostaglandin J2 in Activated Murine Macrophages

Antonio Castrillo, +4 more
- 01 Mar 2000 - 
TL;DR: In this paper, 15-deoxy-Δ12,14-prostaglandin J2 (15dPGJ2) was shown to inhibit the degradation of IκBα and increased their levels in the nuclei.