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Shigeru Oshima

Researcher at Tokyo Medical and Dental University

Publications -  66
Citations -  8821

Shigeru Oshima is an academic researcher from Tokyo Medical and Dental University. The author has contributed to research in topics: Colitis & Tumor necrosis factor alpha. The author has an hindex of 27, co-authored 63 publications receiving 7184 citations. Previous affiliations of Shigeru Oshima include University of California, San Francisco.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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CD4+CD25bright T Cells in Human Intestinal Lamina Propria as Regulatory Cells

Shin Makita, +9 more
- 01 Sep 2004 - 
TL;DR: The results suggest that CD4+CD25bright T cells found in the normal and inflamed intestinal mucosa selectively inhibit the host immune response and therefore may contribute to the intestinal immune homeostasis.
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Expression of A20 by dendritic cells preserves immune homeostasis and prevents colitis and spondyloarthritis

Gianna E. Hammer, +17 more
- 01 Dec 2011 - 
TL;DR: It is shown that mice lacking the ubiquitin-editing molecule A20 specifically in DCs spontaneously showed DC activation and population expansion of activated T cells, and suggested that A20-dependent DC functions may underlie IBD and IBD-associated arthritides.
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The ubiquitin-modifying enzyme A20 restricts ubiquitination of the kinase RIPK3 and protects cells from necroptosis

Michio Onizawa, +17 more
- 01 Jun 2015 - 
TL;DR: This work has found that A20-deficient T cells and fibroblasts were susceptible to caspase-independent and kinase RIPK3–dependent necroptosis and suggested additional mechanisms by which A20 might prevent inflammatory disease.