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Avnika A. Ruparelia

Researcher at Monash University

Publications -  21
Citations -  6844

Avnika A. Ruparelia is an academic researcher from Monash University. The author has contributed to research in topics: Skeletal muscle & Myopathy. The author has an hindex of 11, co-authored 20 publications receiving 5446 citations. Previous affiliations of Avnika A. Ruparelia include Australian Regenerative Medicine Institute.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Filamin C is a highly dynamic protein associated with fast repair of myofibrillar microdamage

Yvonne Leber, +8 more
- 19 May 2016 - 
TL;DR: It is demonstrated by fluorescence recovery after photobleaching that a large fraction of FLNc is highly mobile in cultured neonatal mouse cardiomyocytes and in cardiac and skeletal muscles of live transgenic zebrafish embryos and this results help to better understand the pathomechanisms and pathophysiology of early stages ofFLNc-related myofibrillar myopathy and skeletal and cardiac diseases preceding pathological protein aggregation.
Journal ArticleDOI

Zebrafish models of BAG3 myofibrillar myopathy suggest a toxic gain of function leading to BAG3 insufficiency

Avnika A. Ruparelia, +4 more
- 02 Oct 2014 - 
TL;DR: Together, knockdown and overexpression experiments identify a mechanism whereby BAG3P209L aggregates form, gradually reducing the pool of available B AG3, which eventually results in BAG 3 insufficiency and myofibrillar disintegration, which suggests that reducing aggregation through enhanced degradation or inhibition of nucleation would be an effective therapy for this disease.