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Fatima Teixeira-Clerc

Researcher at University of Paris

Publications -  36
Citations -  9904

Fatima Teixeira-Clerc is an academic researcher from University of Paris. The author has contributed to research in topics: Cannabinoid receptor type 2 & Cannabinoid receptor. The author has an hindex of 23, co-authored 35 publications receiving 8180 citations. Previous affiliations of Fatima Teixeira-Clerc include Paris 12 Val de Marne University & French Institute of Health and Medical Research.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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CB1 cannabinoid receptor antagonism: a new strategy for the treatment of liver fibrosis.

Fatima Teixeira-Clerc, +17 more
- 21 May 2006 - 
TL;DR: The study shows that CB1 receptor antagonists hold promise for the treatment of liver fibrosis, and suggests that endocannabinoids may drive both CB2-mediated antifibrogenic effects andCB2-independent profibrogenics effects.
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Antifibrogenic role of the cannabinoid receptor CB2 in the liver.

Boris Julien, +15 more
- 01 Mar 2005 - 
TL;DR: These data constitute the first demonstration that CB2 receptors are highly up-regulated in the cirrhotic liver, predominantly in hepatic fibrogenic cells and highlight the antifibrogenic role of CB2 receptor during chronic liver injury.
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M2 Kupffer cells promote M1 Kupffer cell apoptosis: a protective mechanism against alcoholic and nonalcoholic fatty liver disease.

Jinghong Wan, +11 more
- 01 Jan 2014 - 
TL;DR: It is suggested that promoting M2‐induced M1 KC apoptosis might prove a relevant strategy to limit alcohol‐ and high fat‐induced inflammation and hepatocyte injury.