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Galila Agam

Researcher at Ben-Gurion University of the Negev

Publications -  183
Citations -  11297

Galila Agam is an academic researcher from Ben-Gurion University of the Negev. The author has contributed to research in topics: Lithium (medication) & Inositol. The author has an hindex of 38, co-authored 180 publications receiving 9630 citations. Previous affiliations of Galila Agam include Soroka Medical Center & Stanley Foundation.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

A CSF and postmortem brain study of D-serine metabolic parameters in schizophrenia.

TL;DR: The results suggest that reduced brain serine racemase and elevated D-amino acid oxidase protein levels may contribute to the lower CSF D-serine levels in schizophrenia.
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Autophagy has a key role in the pathophysiology of schizophrenia.

TL;DR: A brain-specific reduction in beclin1 expression is shown in postmortem hippocampus of schizophrenia patients, not detected in peripheral lymphocytes, suggesting a direct association with the autophagy process and paving the path to novel targets for drug design.
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Low GSK-3beta immunoreactivity in postmortem frontal cortex of schizophrenic patients.

TL;DR: Results are consistent with the notion that schizophrenia involves neurodevelopmental pathology, and it remains to be investigated whether the active fraction of GSK-3beta is also low in frontal cortex of schizophrenic patients and if this is also reflected in other brain regions.