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Jorrit M. Enserink

Researcher at University of Oslo

Publications -  71
Citations -  9333

Jorrit M. Enserink is an academic researcher from University of Oslo. The author has contributed to research in topics: Cyclin-dependent kinase 1 & Cyclin-dependent kinase. The author has an hindex of 25, co-authored 58 publications receiving 7760 citations. Previous affiliations of Jorrit M. Enserink include University of California, San Diego & University of Massachusetts Medical School.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

A novel Epac-specific cAMP analogue demonstrates independent regulation of Rap1 and ERK.

Jorrit M. Enserink, +8 more
- 01 Nov 2002 - 
TL;DR: A novel cAMP analogue is developed, 8CPT-2Me-cAMP, which activates Epac, but not PKA, both in vitro and in vivo, and tests the widespread model that Rap1 mediates cAMP-induced regulation of the extracellular signal-regulated kinase (ERK).
Journal ArticleDOI

An overview of Cdk1-controlled targets and processes

Jorrit M. Enserink, +1 more
- 13 May 2010 - 
TL;DR: Currently known targets of Cdk1 in the budding yeast S. cerevisiae are discussed and the role of Ctk1 in several crucial processes including maintenance of genome stability is highlighted.
Journal ArticleDOI

Cyclic AMP induces integrin-mediated cell adhesion through Epac and Rap1 upon stimulation of the β2-adrenergic receptor

Savithri Rangarajan, +6 more
- 17 Feb 2003 - 
TL;DR: It is demonstrated that external stimuli of cAMP signaling, i.e., isoproterenol, which activates the Gαs-coupled β2-adrenergic receptor can induce integrin-mediated cell adhesion through the Epac-Rap1 pathway.