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Shohreh Issazadeh-Navikas

Researcher at University of Copenhagen

Publications -  63
Citations -  8825

Shohreh Issazadeh-Navikas is an academic researcher from University of Copenhagen. The author has contributed to research in topics: T cell & Natural killer T cell. The author has an hindex of 23, co-authored 63 publications receiving 7228 citations. Previous affiliations of Shohreh Issazadeh-Navikas include Lund University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Neuron-mediated generation of regulatory T cells from encephalitogenic T cells suppresses EAE.

Yawei Liu, +3 more
- 23 Apr 2006 - 
TL;DR: Findings show that generation of neuron-dependent Treg cells in the CNS is instrumental in regulating CNS inflammation, having a crucial role in governing T-cell response and central nervous system (CNS) inflammation.
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Dual effects of vitamin D–induced alteration of TH1/TH2 cytokine expression: Enhancing IgE production and decreasing airway eosinophilia in murine allergic airway disease

Victor Matheu, +3 more
- 01 Sep 2003 - 
TL;DR: It is suggested that excess supplementation of vitamin D could influence the development of a sustained T(H)2 response, leading to an increasing prevalence of allergy, whereas vitamin D might hold promising beneficial effects in airway eosinophilia.
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IFN-beta gene deletion leads to augmented and chronic demyelinating experimental autoimmune encephalomyelitis.

Ingrid Teige, +7 more
- 01 May 2003 - 
TL;DR: It is suggested that lack of endogenous IFN-β in CNS leads to augmented microglia activation, resulting in a sustained inflammation, cytokine production, and tissue damage with consequent chronic neurological deficits.