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Bo Wang

Researcher at Tongji University

Publications -  44
Citations -  8003

Bo Wang is an academic researcher from Tongji University. The author has contributed to research in topics: Medicine & Peroxisome. The author has an hindex of 16, co-authored 30 publications receiving 6273 citations. Previous affiliations of Bo Wang include University of Michigan & Guangxi Medical University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Role of peroxisomes in ROS/RNS-metabolism: implications for human disease.

TL;DR: Evidence supporting the notion that peroxisomal metabolism and oxidative stress are intimately interconnected and associated with age-related diseases is discussed.
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SHMT2 Desuccinylation by SIRT5 Drives Cancer Cell Proliferation

TL;DR: The results reveal that SHMT2 desuccinylation is a pivotal signal in cancer cells to adapt serine metabolic processes for rapid growth, and they highlight SIRT5 as a candidate target for suppressing serine catabolism as a strategy to block tumor growth.
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MicroRNA-21 prevents excessive inflammation and cardiac dysfunction after myocardial infarction through targeting KBTBD7.

TL;DR: It is demonstrated that miR-21 attenuates inflammation, cardiac dysfunction, and maladaptive remodeling post MI through targeting KBTBD7 and inhibiting p38 and NF-κB signaling activation, suggesting that mi R-21 may function as a novel potential therapeutic target for MI.