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Soo Han Bae

Researcher at Yonsei University

Publications -  58
Citations -  9813

Soo Han Bae is an academic researcher from Yonsei University. The author has contributed to research in topics: Autophagy & Sulfiredoxin. The author has an hindex of 27, co-authored 53 publications receiving 7922 citations. Previous affiliations of Soo Han Bae include University of Texas Southwestern Medical Center & Ewha Womans University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Peroxiredoxin Functions as a Peroxidase and a Regulator and Sensor of Local Peroxides

Sue Goo Rhee, +3 more
- 10 Feb 2012 - 
TL;DR: Peroxiredoxins contain an active site cysteine that is sensitive to oxidation by H2O2, and Regulation of Prx via phosphorylation in response to extracellular signals allows the local accumulation of H2 O2 and thereby enables its messenger function.
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Sestrins Activate Nrf2 by Promoting p62-Dependent Autophagic Degradation of Keap1 and Prevent Oxidative Liver Damage

Soo Han Bae, +8 more
- 08 Jan 2013 - 
TL;DR: Sesn1 and Sesn2 interact with the Nrf2 suppressor Keap1, the autophagy substrate p62, and the ubiquitin ligase Rbx1 and it is shown that the antioxidant function of Sesns is mediated through activation of NRF2 in a manner reliant on p62-dependent autophagic degradation of Keap 1.
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Insulin stimulation of SREBP-1c processing in transgenic rat hepatocytes requires p70 S6-kinase

Joshua L. Owen, +8 more
- 02 Oct 2012 - 
TL;DR: The data indicate that the pathways for insulin enhancement of SREBP-1c mRNA and proteolytic processing diverge after mTORC1, and the transgenic rat system will be useful in further defining the molecular mechanism for insulin stimulation of lipid synthesis in liver in normal and diabetic states.