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Richard W. Wong

Researcher at Kanazawa University

Publications -  51
Citations -  7862

Richard W. Wong is an academic researcher from Kanazawa University. The author has contributed to research in topics: Nuclear pore & Nucleoporin. The author has an hindex of 25, co-authored 51 publications receiving 6280 citations. Previous affiliations of Richard W. Wong include Howard Hughes Medical Institute.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Rae1 interaction with NuMA is required for bipolar spindle formation

TL;DR: The Rae1–NuMA interaction is identified as a critical element for normal spindle formation in mitosis and a specific binding site for Rae1 on NuMA is mapped that would convert a NuMA dimer to a “tetravalent” crosslinker of MTs.
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Structural and functional analysis of the interaction between the nucleoporin Nup214 and the DEAD-box helicase Ddx19.

TL;DR: It is speculated that the positively charged surface of the interacting ADP-helicase binds competitively to a segment of mRNA of a linearized mRNP, passing through the NPC on its way to the cytoplasm, and would replace a single bound protein from the mRNA.
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ZNF750 is a lineage-specific tumour suppressor in squamous cell carcinoma.

TL;DR: It is reported that ZNF 750 is exclusively deleted, mutated and underexpressed in human SCCs, and low ZNF750 expression is associated with poor survival, and its novel anticancer-associated functions are uncovered.