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Eliana M. Coccia

Researcher at Istituto Superiore di Sanità

Publications -  123
Citations -  12580

Eliana M. Coccia is an academic researcher from Istituto Superiore di Sanità. The author has contributed to research in topics: Interferon & Immune system. The author has an hindex of 43, co-authored 123 publications receiving 10612 citations. Previous affiliations of Eliana M. Coccia include Marche Polytechnic University & Sapienza University of Rome.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Infection of Human Macrophages and Dendritic Cells with Mycobacterium tuberculosis Induces a Differential Cytokine Gene Expression That Modulates T Cell Response

TL;DR: Analysis of early interactions between Mycobacterium tuberculosis and immune cells suggested that IL-12 and, to a lesser extent, IFN-α and IL-18 play a significant role in enhancingIFN-γ synthesis by T cells.
Journal ArticleDOI

ESX-1 dependent impairment of autophagic flux by Mycobacterium tuberculosis in human dendritic cells

TL;DR: It is demonstrated that Mtb alters the autophagic machinery through the ESX-1 system, and thereby opens new exciting perspectives to better understand the relationship between Mtb virulence and its ability to escape the DC-mediated immune response.