scispace - formally typeset
S

Satoaki Matoba

Researcher at Kyoto Prefectural University of Medicine

Publications -  325
Citations -  13747

Satoaki Matoba is an academic researcher from Kyoto Prefectural University of Medicine. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 41, co-authored 254 publications receiving 11260 citations. Previous affiliations of Satoaki Matoba include Kyoto Prefectural University & National Institutes of Health.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

p53 Regulates Mitochondrial Respiration

Satoaki Matoba, +16 more
- 16 Jun 2006 - 
TL;DR: It is shown that p53, one of the most frequently mutated genes in cancers, modulates the balance between the utilization of respiratory and glycolytic pathways, and Synthesis of Cytochrome c Oxidase 2 (SCO2) is identified as the downstream mediator of this effect.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Cytosolic p53 inhibits Parkin-mediated mitophagy and promotes mitochondrial dysfunction in the mouse heart

Atsushi Hoshino, +9 more
- 06 Aug 2013 - 
TL;DR: It is shown that cytosolic p53 binds to Parkin and disturbs its translocation to damaged mitochondria and their subsequent clearance by mitophagy and overexpression of Parkin ameliorates the functional decline in aged hearts, and is accompanied by decreased senescence-associated β-galactosidase activity and proinflammatory phenotypes.
Journal ArticleDOI

Long-term clinical outcome after intramuscular implantation of bone marrow mononuclear cells (Therapeutic Angiogenesis by Cell Transplantation [TACT] trial) in patients with chronic limb ischemia

Satoaki Matoba, +17 more
- 01 Nov 2008 - 
TL;DR: The angiogenic cell therapy using bone marrow mononuclear cells can induce a long-term improvement in limb ischemia, leading to extension of amputation-free interval, and the safety and efficacy are not inferior to the conventional revascularization therapies.