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Núria S. Coll

Researcher at Spanish National Research Council

Publications -  64
Citations -  9523

Núria S. Coll is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: Ralstonia solanacearum & Programmed cell death. The author has an hindex of 24, co-authored 53 publications receiving 7511 citations. Previous affiliations of Núria S. Coll include ETH Zurich & University of Barcelona.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Programmed cell death in the plant immune system

TL;DR: This review describes the molecular pathways leading to cell death during innate immune responses and presents recently discovered caspase andcaspase-like networks regulating cell death that have revealed fascinating analogies between cell death control across both kingdoms.
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Arabidopsis Type I Metacaspases Control Cell Death

TL;DR: Manipulation of the Arabidopsis type I metacaspase regulatory module can nearly eliminate the hypersensitive cell death response activated by plant intracellular immune receptors, but does not lead to enhanced pathogen proliferation, decoupling HR from restriction of pathogen growth.
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Plant innate immunity – sunny side up?

TL;DR: It is proposed that this chloroplastic branch acts as a light-dependent rheostat that, through the production of ROS, influences the severity of the immune response.