scispace - formally typeset
C

Claudio Luparello

Researcher at University of Palermo

Publications -  87
Citations -  8212

Claudio Luparello is an academic researcher from University of Palermo. The author has contributed to research in topics: Gene expression & Apoptosis. The author has an hindex of 23, co-authored 79 publications receiving 6684 citations.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Cadmium and mitochondria.

TL;DR: This review provides new additional informations on the cellular and molecular aspects of the interaction between Cd and cells, emphasizing alterations of mitochondria as important events in Cd cytotoxicity, thus representing an important basis for understanding the mechanisms of cadmium effect on the cells.
Journal ArticleDOI

Purinergic receptors influence the differentiation of human mesenchymal stem cells.

TL;DR: It is shown for the first time that the combination of several P2 receptors plays a role in the differentiation of adult stem cells and might reveal novel options to influence stem cell fate in future applications.
Journal ArticleDOI

Tissue inhibitor of metalloprotease (TIMP)-1 and proliferative behaviour of clonal breast cancer cells.

TL;DR: The hypothesis that hTIMP‐1, ‘classically’ regarded as a collagenase inhibitor, may be a crucial element of the extracellular signalling network during breast cancer development by controlling cell growth phenotype in autocrine and paracrine manner is supported.