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Maria F. Galindo

Researcher at University of Castilla–La Mancha

Publications -  71
Citations -  9661

Maria F. Galindo is an academic researcher from University of Castilla–La Mancha. The author has contributed to research in topics: Mitochondrion & Programmed cell death. The author has an hindex of 36, co-authored 66 publications receiving 8113 citations. Previous affiliations of Maria F. Galindo include Universidad Miguel Hernández de Elche & University of Chicago.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Isoform-specific effect of apolipoprotein E on cell survival and β- amyloid-induced toxicity in rat hippocampal pyramidal neuronal cultures

Joaquín Jordán, +5 more
- 01 Jan 1998 - 
TL;DR: The protection from Aβ-induced neurotoxicity afforded by apoE3 treatment may result from clearance of the peptide by apOE3:Aβ complex formation and uptake by apolipoprotein E receptors.
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Role of Calpain‐ and Interleukin‐1β Converting Enzyme‐Like Proteases in the β‐Amyloid‐Induced Death of Rat Hippocampal Neurons in Culture

Joaquín Jordán, +2 more
- 18 Nov 2002 - 
TL;DR: The results support the role of apoptosis in neuronal death due to Aβ(25–35) treatment and also suggest a role for calcium‐regulated proteases in this process.
Journal Article

Protective effect of transforming growth factor-beta 1 on beta-amyloid neurotoxicity in rat hippocampal neurons.

Jochen H. M. Prehn, +10 more
- 01 Feb 1996 - 
TL;DR: It is found that A beta neurotoxicity was significantly attenuated by single treatments with TGF-beta 1 and prevented by repetitive treatments, and the protective effects of TGF -beta 1 were associated with a preservation of mitochondrial potential and function, as determined with rhodamine-123-based microfluorimetry.