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Manjula Kalia

Researcher at Translational Health Science and Technology Institute

Publications -  40
Citations -  7715

Manjula Kalia is an academic researcher from Translational Health Science and Technology Institute. The author has contributed to research in topics: Virus & Autophagy. The author has an hindex of 16, co-authored 31 publications receiving 6189 citations. Previous affiliations of Manjula Kalia include International Centre for Genetic Engineering and Biotechnology & National Centre for Biological Sciences.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Molecular biology and pathogenesis of hepatitis E virus

Vivek Chandra, +3 more
- 24 Dec 2008 - 
TL;DR: This review describes HEV epidemiology, clinical presentation, pathogenesis, molecular virology and the host response to HEV infection in published literature in the past decade.
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Heparan Sulfate Proteoglycans Are Required for Cellular Binding of the Hepatitis E Virus ORF2 Capsid Protein and for Viral Infection

Manjula Kalia, +4 more
- 15 Dec 2009 - 
TL;DR: It is demonstrated that cell surface heparan sulfate proteoglycans (HSPGs), specifically syndecans, play a crucial role in the binding of pORF2 to Huh-7 liver cells, indicating that a nonenveloped virus like HEV may have also evolved to use HSPGs as cellular attachment receptors.
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Japanese Encephalitis Virus Infects Neuronal Cells through a Clathrin-Independent Endocytic Mechanism

Manjula Kalia, +4 more
- 01 Jan 2013 - 
TL;DR: It is demonstrated that JEV infects fibroblasts in a clathrin-dependent manner, but it deploys a clATHrin-independent mechanism to infect neuronal cells, which requires dynamin and plasma membrane cholesterol.