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Marta Margeta

Researcher at University of California, San Francisco

Publications -  43
Citations -  7478

Marta Margeta is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Myopathy & Autophagy. The author has an hindex of 17, co-authored 38 publications receiving 5993 citations. Previous affiliations of Marta Margeta include University of California & University of California, San Diego.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Regulatory T cells suppress muscle inflammation and injury in muscular dystrophy

TL;DR: The findings suggest that Tregs modulate the progression of muscular dystrophy by suppressing type 1 inflammation in muscle associated with muscle fiber injury, and highlight the potential of Treg-modulating agents as therapeutics for DMD.
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Complex formation with the Type B gamma-aminobutyric acid receptor affects the expression and signal transduction of the extracellular calcium-sensing receptor. Studies with HEK-293 cells and neurons.

TL;DR: CaRs and GABA-B-R subunits can form heteromeric complexes in cells, and their interactions affect cell surface expression and signaling of CaR, which may contribute to extracellular Ca2+-dependent receptor activation in target tissues.
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Rare somatic cells from human breast tissue exhibit extensive lineage plasticity.

TL;DR: This subpopulation of cells from healthy human breast tissue is poised to transcribe plasticity markers, OCT3/4, SOX2, and NANOG, at levels similar to those measured in human embryonic stem cells and to acquire a plastic state sensitive to environmental programming.