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Oliver Florey

Researcher at Babraham Institute

Publications -  54
Citations -  9989

Oliver Florey is an academic researcher from Babraham Institute. The author has contributed to research in topics: Autophagy & ATG8. The author has an hindex of 26, co-authored 50 publications receiving 7914 citations. Previous affiliations of Oliver Florey include Memorial Sloan Kettering Cancer Center & Imperial College London.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Autophagy machinery mediates macroendocytic processing and entotic cell death by targeting single membranes

Oliver Florey, +7 more
- 01 Nov 2011 - 
TL;DR: It is demonstrated that proteins of the autophagy pathway can target single-membrane vacuoles in cells in the absence of pathogenic organisms.
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Proinflammatory activation of macrophages by basic calcium phosphate crystals via protein kinase C and MAP kinase pathways : A vicious cycle of inflammation and arterial calcification?

Imad Nadra, +7 more
- 24 Jun 2005 - 
TL;DR: The response of macrophages to BCP crystals suggests that pathological calcification is not merely a passive consequence of chronic inflammatory disease but may lead to a positive feed-back loop of calcification and inflammation driving disease progression.
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TLR Signals Induce Phagosomal MHC-I Delivery from the Endosomal Recycling Compartment to Allow Cross-Presentation

Priyanka Nair-Gupta, +13 more
- 31 Jul 2014 - 
TL;DR: It is shown that MHC-I selectively accumulate within phagosomes carrying microbial components, which engage Toll-like receptor (TLR) signaling and subsequent cross-presentation during infection.