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Alkesh Jani

Researcher at Veterans Health Administration

Publications -  50
Citations -  7694

Alkesh Jani is an academic researcher from Veterans Health Administration. The author has contributed to research in topics: Transplantation & Acute kidney injury. The author has an hindex of 23, co-authored 47 publications receiving 7065 citations. Previous affiliations of Alkesh Jani include Stanford University & Anschutz Medical Campus.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Urinary interleukin-18 is a marker of human acute tubular necrosis.

Chirag R. Parikh, +4 more
- 01 Mar 2004 - 
TL;DR: In this paper, the IL-18 was measured in human urine to determine whether it might serve as a marker of acute tubular necrosis (ATN) in mice, including healthy controls, patients with different forms of acute renal failure, and patients with other renal diseases.
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Urine NGAL and IL-18 are predictive biomarkers for delayed graft function following kidney transplantation.

Chirag R. Parikh, +7 more
- 01 Jul 2006 - 
TL;DR: Results indicate that urinary neutrophil gelatinase‐associated lipocalin and interleukin‐18 represent early, predictive biomarkers of DGF, and both on day 0 predicted the trend in serum creatinine in the posttransplant period.
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IL-33 Exacerbates Acute Kidney Injury

Ali Akcay, +12 more
- 01 Nov 2011 - 
TL;DR: Increased protein expression of full-length IL-33 in the kidney following induction of AKI with cisplatin and higher levels of the proinflammatory chemokine CXCL1, which CD T cells produce, are observed in wildtype mice compared with CD4-deficient mice, suggesting that inhibiting IL- 33 or CX CL1 may have therapeutic potential in AKI.
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NLRP3 Inflammasome Knockout Mice Are Protected against Ischemic but Not Cisplatin-Induced Acute Kidney Injury

Hyun-Jung Kim, +9 more
- 01 Sep 2013 - 
TL;DR: The difference in protection against cisplatin-induced AKI compared with ischemic AKI in NLRP3−/− mice was not explained by the differences in proinflammatory cytokines interleukin (IL)-1β, IL-6, chemokine (C-X-C motif) ligand 1, or tumor necrosis factor α.