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Farida V. Minibayeva

Researcher at Russian Academy of Sciences

Publications -  98
Citations -  8608

Farida V. Minibayeva is an academic researcher from Russian Academy of Sciences. The author has contributed to research in topics: Lichen & Peroxidase. The author has an hindex of 26, co-authored 80 publications receiving 7778 citations. Previous affiliations of Farida V. Minibayeva include Russian Academy & Royal Holloway, University of London.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The apoplastic oxidative burst in response to biotic stress in plants: a three‐component system

TL;DR: The oxidative burst, the generation of reactive oxygen species (ROS) in response to microbial pathogen attack, is a ubiquitous early part of the resistance mechanisms of plant cells and the mechanism has proved to be complex and may involve a number of low molecular weight components.
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What is stress? Concepts, definitions and applications in seed science.

TL;DR: The 'alarm' response is defined by post-translational modifications and stress signalling through cross-talk between reactive oxygen and nitrogen species, and seed hormones, that result in modifications to the transcriptome, corresponding to 'exhaustion'.
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Plant sterols: Diversity, biosynthesis, and physiological functions

TL;DR: This review focuses on the key stages of plant sterol biosynthesis that determine the ratios between the different types of sterols, and the crosstalk between the sterol and sphingolipid pathways.
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Early signalling events in the apoplastic oxidative burst in suspension cultured French bean cells involve cAMP and Ca2

TL;DR: An increase of cytosolic calcium mediated through an increased level of cAMP is required for ROS production, which supports a role for G proteins and cAMP in extracellular alkalinization and Ca2+ influx, possibly in the provision of a reductant, which with the Extracellular peroxidase, are required for the apoplastic oxidative burst.