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Fei Liu

Researcher at University of Michigan

Publications -  54
Citations -  7469

Fei Liu is an academic researcher from University of Michigan. The author has contributed to research in topics: Autophagy & Osteoblast. The author has an hindex of 22, co-authored 42 publications receiving 6575 citations. Previous affiliations of Fei Liu include China Medical University (PRC) & University of Connecticut Health Center.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Autophagy in stem cells

TL;DR: A comprehensive review of the current understanding of the mechanisms and regulation of autophagy in embryonic stem cells, several tissue stem cells (particularly hematopoietic stem cells), as well as a number of cancer stem cells is provided.
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NF-κB inhibits osteogenic differentiation of mesenchymal stem cells by promoting β-catenin degradation

TL;DR: The results suggest that targeting IKK–NF-κB may have dual benefits in enhancing bone regeneration and repair and inhibiting inflammation, and this concept may also have applicability in many other tissue regeneration situations.
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FIP200 is required for the cell-autonomous maintenance of fetal hematopoietic stem cells.

TL;DR: It is shown that conditional deletion of FIP200 in hematopoietic cells leads to perinatal lethality and severe anemia and a potential role for autophagy in the maintenance of fetal hematoiesis and HSCs is implicate.
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Expression and activity of osteoblast-targeted Cre recombinase transgenes in murine skeletal tissues.

TL;DR: The authors' data suggest that Col 2.3-Cre and Col 3.6-Cre transgenic mice may be useful for conditional gene targeting in vivo or for obtaining osteoblast populations for in vitro culture in which a gene of interest has been inactivated.