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John H. Fingert

Researcher at University of Iowa

Publications -  171
Citations -  14975

John H. Fingert is an academic researcher from University of Iowa. The author has contributed to research in topics: Glaucoma & Myocilin. The author has an hindex of 47, co-authored 165 publications receiving 13442 citations. Previous affiliations of John H. Fingert include University of Arkansas for Medical Sciences & University of Iowa Hospitals and Clinics.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Identification of a Gene That Causes Primary Open Angle Glaucoma

Edwin M. Stone, +14 more
- 31 Jan 1997 - 
TL;DR: A gene encoding a trabecular meshwork protein (TIGR) mapped to the narrowest disease interval by STS content and radiation hybrid mapping and will aid in early diagnosis of glaucoma.
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Primary open-angle glaucoma.

Young H. Kwon, +3 more
- 10 Dec 2009 - 
TL;DR: A typical case of glaucoma is presented, together with the ocular findings, and the clinical features, genetics, molecular biology, and cell biology of glAUcoma are discussed.
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Analysis of Myocilin Mutations in 1703 Glaucoma Patients From Five Different Populations

John H. Fingert, +19 more
- 01 May 1999 - 
TL;DR: Although the specific mutations found in each of the five populations were different, the overall frequency of myocilin mutations was similar ( approximately 2-4%) in all populations, suggesting that the increased rate of glaucoma in African Americans is not due to a higher prevalence of myOCilin mutation.
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Clinical Features Associated with Mutations in the Chromosome 1 Open-Angle Glaucoma Gene (GLC1A)

Wallace L.M. Alward, +11 more
- 09 Apr 1998 - 
TL;DR: 16 sequence variations that met the criteria for a probable disease-causing mutation because they altered the predicted amino acid sequence were identified and they were found in one or more patients with glaucoma, in less than 1 percent of the control subjects.