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Daniela Trisciuoglio

Researcher at National Research Council

Publications -  89
Citations -  9685

Daniela Trisciuoglio is an academic researcher from National Research Council. The author has contributed to research in topics: Cancer & Apoptosis. The author has an hindex of 35, co-authored 77 publications receiving 8296 citations. Previous affiliations of Daniela Trisciuoglio include Sapienza University of Rome & Science Applications International Corporation.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Apoptosis as anticancer mechanism: function and dysfunction of its modulators and targeted therapeutic strategies

Giuseppa Pistritto, +4 more
TL;DR: The role of the fundamental regulators of apoptosis are highlighted and how their deregulation, including activation of anti-apoptotic factors or inactivation of pro-ap optotic factors ends up in cancer cell resistance to therapies.
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Antiangiogenic potential of the Mammalian target of rapamycin inhibitor temsirolimus.

Donatella Del Bufalo, +6 more
- 01 Jun 2006 - 
TL;DR: Results suggest that antiangiogenic effects may substantially contribute to the antitumor activity observed with temsirolimus in breast cancer.
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The execution of the transcriptional axis mutant p53, E2F1 and ID4 promotes tumor neo-angiogenesis

Giulia Fontemaggi, +12 more
- 27 Sep 2009 - 
TL;DR: The identification of ID4 as a transcriptional target of gain-of-function p53 mutants R175H, R273H and R280K highlights the transcriptional axis mutant p53, E2F1 and ID 4 as a still undefined molecular mechanism contributing to tumor neo-angiogenesis.
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Endothelin-1 protects ovarian carcinoma cells against paclitaxel-induced apoptosis: requirement for Akt activation

Donatella Del Bufalo, +8 more
- 01 Mar 2002 - 
TL;DR: A novel role is established in determining protection of ovarian carcinoma cells against paclitaxel-induced apoptosis through Bcl-2-dependent and PI3-K-mediated Akt pathways and suggest that ET-1 and ET(A)R could represent important targets for anticancer therapy.