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Yide Mei

Researcher at University of Science and Technology of China

Publications -  50
Citations -  7913

Yide Mei is an academic researcher from University of Science and Technology of China. The author has contributed to research in topics: Ubiquitin ligase & Regulation of gene expression. The author has an hindex of 26, co-authored 45 publications receiving 7078 citations. Previous affiliations of Yide Mei include University of Pennsylvania.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Reciprocal Regulation of HIF-1α and LincRNA-p21 Modulates the Warburg Effect

Fan Yang, +3 more
- 09 Jan 2014 - 
TL;DR: It is shown that lincRNA-p21 is a hypoxia-responsive lncRNA and is essential for hypoxIA-enhanced glycolysis and the ability of lincRNAs to promote tumor growth is validated in mouse xenograft models and implicate linc RNA- p21 as a valuable therapeutic target for cancer.
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Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain.

Shanshan Feng, +7 more
- 01 Oct 2007 - 
TL;DR: It is shown that RIP3 is cleaved at Asp328 by caspase-8 under apoptotic stimuli, which is blocked by pan-caspase inhibitor Z-VAD-FMK, and the cleaved product of RIP3 (aa 329-518) displays better stability than wild type RIP3.
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tRNA Binds to Cytochrome c and Inhibits Caspase Activation

Yide Mei, +6 more
- 12 Mar 2010 - 
TL;DR: TRNA hydrolysis in living cells and cell lysates enhances apoptosis and caspase activation, whereas microinjection of tRNA into living cells blocks apoptosis, suggesting that tRNA, in addition to its well-established role in gene expression, may determine cellular responsiveness to apoptotic stimuli.
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XIAP inhibits autophagy via XIAP‐Mdm2‐p53 signalling

Xing Huang, +3 more
- 14 Aug 2013 - 
TL;DR: Findings reveal a novel XIAP‐Mdm2‐p53 pathway that mediates the inhibition of autophagy, by which XIAP may contribute to tumorigenesis.