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Yoshihiro Shidoji

Researcher at Health Science University

Publications -  86
Citations -  6807

Yoshihiro Shidoji is an academic researcher from Health Science University. The author has contributed to research in topics: Retinoic acid & Gene. The author has an hindex of 21, co-authored 84 publications receiving 6274 citations. Previous affiliations of Yoshihiro Shidoji include Siebold University of Nagasaki & RMIT University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Induction of apoptosis by acyclic retinoid in the human hepatoma-derived cell line, HuH-7.

TL;DR: HuH-7 cells, a human hepatoma-derived cell line, underwent apoptosis in response to all-trans 3, 7, 11, 15-tetramethyl- 2, 4, 6, 10, 14-hexadecapentaenoic acid, or acyclic retinoid, indicating that the antitumor activity of the acy Clinoid may be partly explained by the induction of apoptosis.
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Retinoid agonist activities of synthetic geranyl geranoic acid derivatives.

TL;DR: It is shown for the first time that chemically synthesized acyclic organic acids are potential agonists of natural retinoids.
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Analysis of Gene Expression Profile Induced by Hepatocyte Nuclear Factor 4α in Hepatoma Cells Using an Oligonucleotide Microarray

TL;DR: Huang et al. as mentioned in this paper constructed an adenovirus vector carrying rat HNF-4α cDNA and transfected the vector to human hepatoma cells, HuH-7, to enforce expression of the exogenous HNF4α gene.
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Regulation of Hepatic Genes and Liver Transcription Factors in Rat Hepatocytes by Extracellular Matrix

TL;DR: It is suggested that cell-matrix interaction may determine and maintain the differentiated phenotype of hepatocytes by regulating liver-specific transcription factors.