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Robert G. Hawley

Researcher at George Washington University

Publications -  205
Citations -  16465

Robert G. Hawley is an academic researcher from George Washington University. The author has contributed to research in topics: Stem cell & Haematopoiesis. The author has an hindex of 59, co-authored 197 publications receiving 15648 citations. Previous affiliations of Robert G. Hawley include Jining Medical University & Cornell University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Versatile retroviral vectors for potential use in gene therapy.

TL;DR: A set of retroviral vectors whose capacity for high efficiency transduction of functional genes into undifferentiated murine embryonic and haematopoietic cells makes them ideally suited for preclinical studies with murine models and might prove useful in human gene therapy protocols.
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Analysis of gene expression in a complex differentiation hierarchy by global amplification of cDNA from single cells

TL;DR: The results provide the first expression mapping of these genes that distinguishes between progenitors in different commitment states, generate new insights and predictions relevant to mechanism, and introduce a powerful set of tools for unravelling the genetic basis of lineage divergence.
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Inhibition of nuclear hormone receptor activity by calreticulin

TL;DR: It is shown that the 60K protein (p60), purified on a KLGFFKR–Sepharose affinity matrix, and recombinant calreticulin can inhibit the binding of androgen receptor to its hormone-responsive DNA element in a KXFFKR-sequence-specific manner.
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High-level sustained transgene expression in human embryonic stem cells using lentiviral vectors.

TL;DR: The ability to efficiently introduce active transgenes into human ES cells will facilitate gain‐of‐function studies of early developmental processes in the human system and have important implications for the possible future use of gene‐modified human ES Cells in transplantation and tissue regeneration applications.