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Durga Nand Tripathi

Researcher at Baylor College of Medicine

Publications -  60
Citations -  8218

Durga Nand Tripathi is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Comet assay & Autophagy. The author has an hindex of 30, co-authored 58 publications receiving 7289 citations. Previous affiliations of Durga Nand Tripathi include Texas A&M Health Science Center & University of Southern California.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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ATM functions at the peroxisome to induce pexophagy in response to ROS

Jiangwei Zhang, +14 more
- 01 Oct 2015 - 
TL;DR: An important new role is revealed for ATM in metabolism as a sensor of ROS that regulates pexophagy in response to ROS, and ATM signalling activates ULK1 and inhibits mTORC1 to induce autophagy.
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Reactive nitrogen species regulate autophagy through ATM-AMPK-TSC2–mediated suppression of mTORC1

Durga Nand Tripathi, +6 more
- 06 Aug 2013 - 
TL;DR: It is found that nitrosative stress induced by steady-state nitric oxide caused rapid activation of an ATM damage-response pathway leading to downstream signaling by this stress kinase to LKB1 and AMPK kinases, and activation of the TSC tumor suppressor.
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A tuberous sclerosis complex signalling node at the peroxisome regulates mTORC1 and autophagy in response to ROS

Jiangwei Zhang, +17 more
- 01 Oct 2013 - 
TL;DR: A role for the TSC in responding to ROS at the peroxisome is identified, and the perxisome as a signalling organelle involved in regulation of mTORC1 is identified.
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Dual Chromatin and Cytoskeletal Remodeling by SETD2.

In Young Park, +16 more
- 11 Aug 2016 - 
TL;DR: These data now identify SETD2 as a dual-function methyltransferase for both chromatin and the cytoskeleton and show a requirement for methylation in maintenance of genomic stability and the integrity of both the tubulin and histone codes.