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Aurelia Lugea

Researcher at Cedars-Sinai Medical Center

Publications -  101
Citations -  9664

Aurelia Lugea is an academic researcher from Cedars-Sinai Medical Center. The author has contributed to research in topics: Pancreatitis & Pancreas. The author has an hindex of 35, co-authored 94 publications receiving 8507 citations. Previous affiliations of Aurelia Lugea include United States Department of Veterans Affairs & University of New South Wales.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The pancreatic stellate cell: a star on the rise in pancreatic diseases.

M. Bishr Omary, +3 more
- 02 Jan 2007 - 
TL;DR: Sustained activation of PaSCs has an increasingly appreciated role in the fibrosis that is associated with chronic pancreatitis and with pancreatic cancer, and offers potential therapeutic targets for the treatment and prevention of these diseases.
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A Starring Role for Stellate Cells in the Pancreatic Cancer Microenvironment

Minoti V. Apte, +6 more
- 01 May 2013 - 
TL;DR: The background and recent advances in pancreatic cancer research began to change with the identification of pancreatic stellate cells, which produce the pancreatic tumor stroma, along with important areas of future research that could improve therapy.
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Cell death in pancreatitis: caspases protect from necrotizing pancreatitis.

Olga A. Mareninova, +7 more
- 10 Feb 2006 - 
TL;DR: The results indicate key roles for caspases, XIAP, and RIP in the regulation of cell death in pancreatitis, and suggest Manipulating these signals to change the pattern of death responses presents a therapeutic strategy for treatment of pancreatitis.
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Desmoplasia of pancreatic ductal adenocarcinoma.

Stephen J. Pandol, +4 more
- 01 Nov 2009 - 
TL;DR: This work has shown that interactions of ECM proteins and desmoplastic secreted growth factors with the cancer cells of PDAC activate intracellular signals including reactive oxygen species that act to make thecancer cells resistant to dying.