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Silvia Alvarez

Researcher at University of Buenos Aires

Publications -  62
Citations -  7162

Silvia Alvarez is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Mitochondrion & Oxidative stress. The author has an hindex of 27, co-authored 56 publications receiving 6491 citations. Previous affiliations of Silvia Alvarez include National Scientific and Technical Research Council.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

The role of mitochondrial nitric oxide synthase in inflammation and septic shock

Alberto Boveris, +2 more
- 01 Nov 2002 - 
TL;DR: The current hypothesis for the molecular mechanisms of septic shock is that the enhanced NO production by mitochondrial nitric oxide synthase leads to excessive peroxynitrite production and protein nitration in the mitochondrial matrix, to mitochondrial dysfunction and to contractile failure.
Journal ArticleDOI

Mitochondrial Mechanisms in Septic Cardiomyopathy

Maria C. Cimolai, +3 more
- 03 Aug 2015 - 
TL;DR: Mitochondrial mechanisms that may causally contribute to mitochondrial dysfunction and/or may represent adaptive responses to mitochondria dysfunction in septic cardiomyopathy are discussed.
Book ChapterDOI

Pharmacological regulation of mitochondrial nitric oxide synthase.

Alberto Boveris, +6 more
- 01 Jan 2002 - 
Journal ArticleDOI

Oxygen dependence of mitochondrial nitric oxide synthase activity

Silvia Alvarez, +3 more
- 06 Jun 2003 - 
TL;DR: The production of NO by mtNOS appears as a regulatory process that modulates mitochondrial oxygen uptake and cellular energy production that correspond to physiological inhibitions of cytochrome oxidase by intramitochondrial NO of 16-25%.