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Péter Nagy

Researcher at Budapest University of Technology and Economics

Publications -  94
Citations -  9005

Péter Nagy is an academic researcher from Budapest University of Technology and Economics. The author has contributed to research in topics: Autophagy & Chemistry. The author has an hindex of 31, co-authored 82 publications receiving 7686 citations. Previous affiliations of Péter Nagy include Hungarian Academy of Sciences & University of Pécs.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Autophagosome-lysosome fusion is independent of V-ATPase-mediated acidification.

TL;DR: It is shown that loss of V-ATPase subunits in the Drosophila fat body causes an accumulation of non-functional lysosomes, leading to a block in autophagic flux, and that BafilomycinA1 inhibits fusion independent of its effect on lysOSomal pH.
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Autophagosomal Syntaxin17-dependent lysosomal degradation maintains neuronal function in Drosophila

TL;DR: Lysosomal degradation and recycling of sequestered autophagosome content is crucial to maintain proper functioning of the fly nervous system.
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The MRCC program system: Accurate quantum chemistry from water to proteins.

TL;DR: The highly competitive linear-scaling local correlation schemes, allow for MP2, RPA, ADC(2), CCSD(T), and higher-order CC calculations for extended systems, and a collection of multi-reference CC and CI approaches are offered.
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Interaction of the HOPS complex with Syntaxin 17 mediates autophagosome clearance in Drosophila

TL;DR: Interaction of the autophagosomal SNARE Syntaxin 17 (Syx17) with the homotypic fusion and vacuole protein–sorting (HOPS) tethering complex is necessary for the fusion of Autophagosomes with lysosomes.