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Daniele Lettieri Barbato

Researcher at University of Rome Tor Vergata

Publications -  16
Citations -  5691

Daniele Lettieri Barbato is an academic researcher from University of Rome Tor Vergata. The author has contributed to research in topics: Adipose tissue & Mitochondrial biogenesis. The author has an hindex of 14, co-authored 15 publications receiving 5182 citations.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

The role of nNOS and PGC-1α in skeletal muscle cells

Sara Baldelli, +4 more
- 15 Nov 2014 - 
TL;DR: Important metabolic pathways that are governed by nNOS and PGC-1&agr; are reviewed, and how they might intersect and cooperatively regulate skeletal muscle mitochondrial and lipid energetic metabolism and contraction is highlighted.
Journal ArticleDOI

Metformin Protects Skeletal Muscle from Cardiotoxin Induced Degeneration

Francesca Langone, +9 more
- 02 Dec 2014 - 
TL;DR: A correlation is reported between the ability of metformin to promote resistance to damage and its capacity to counteract the increment of intracellular calcium levels induced by cardiotoxin treatment, which could prove a valuable strategy to ameliorate the conditions of patients affected by dystrophies.