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Ronit Shiri-Sverdlov

Researcher at Maastricht University

Publications -  118
Citations -  9730

Ronit Shiri-Sverdlov is an academic researcher from Maastricht University. The author has contributed to research in topics: Steatohepatitis & Inflammation. The author has an hindex of 36, co-authored 112 publications receiving 8507 citations. Previous affiliations of Ronit Shiri-Sverdlov include Sheba Medical Center & Tel Aviv University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Dietary cholesterol, rather than liver steatosis, leads to hepatic inflammation in hyperlipidemic mouse models of nonalcoholic steatohepatitis

Kristiaan Wouters, +12 more
- 01 Aug 2008 - 
TL;DR: Findings indicate that dietary cholesterol, possibly in the form of modified plasma lipoproteins, is an important risk factor for the progression to hepatic inflammation in diet‐induced NASH.
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Early diet-induced non-alcoholic steatohepatitis in APOE2 knock-in mice and its prevention by fibrates.

Ronit Shiri-Sverdlov, +9 more
- 01 Apr 2006 - 
TL;DR: Data indicate that inflammation might play an instrumental role during the development of NASH in this mouse model, and fenofibrate treatment decreased hepatic macrophage accumulation and abolished steatosis.
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The Ins and Outs of Cathepsins: Physiological Function and Role in Disease Management.

Tulasi Yadati, +3 more
- 13 Jul 2020 - 
TL;DR: The available evidence regarding the role of cathepsins in health and disease is summarized, their potential as biomarkers of disease progression is discussed, and light is shed on the potential of extracellularCathepsin inhibitors as safe therapeutic tools.
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LDL receptor knock-out mice are a physiological model particularly vulnerable to study the onset of inflammation in non-alcoholic fatty liver disease.

Veerle Bieghs, +11 more
- 25 Jan 2012 - 
TL;DR: The Ldlr−/− mice are a promising physiological model particularly vulnerable for investigating the onset of hepatic inflammation in non-alcoholic steatohepatitis and developed sustained liver damage upon long term HFC feeding due to increased sensitivity for oxLDL uptake.