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Jeremy Thorner

Researcher at University of California, Berkeley

Publications -  237
Citations -  31373

Jeremy Thorner is an academic researcher from University of California, Berkeley. The author has contributed to research in topics: Saccharomyces cerevisiae & Protein kinase A. The author has an hindex of 87, co-authored 234 publications receiving 29999 citations. Previous affiliations of Jeremy Thorner include Stanford University & Oregon Health & Science University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Model systems for the study of seven-transmembrane-segment receptors.

TL;DR: This paper presents a meta-analyses of the chiral signaling process and its applications in medicine and animal welfare, and investigates the role of chiral reprograming in the development of Alzheimer's disease.
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Isolation of the putative structural gene for the lysine-arginine-cleaving endopeptidase required for processing of yeast prepro-α-factor

TL;DR: Gene dosage effects suggest that KEX2 is the structural gene for the endopeptidase, and cloned DNA restores both enzymatic activity in vitro and the normal pattern of proteolytic processing and glycosylation of prepro-alpha-factor in vivo.
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Function and regulation in MAPK signaling pathways: Lessons learned from the yeast Saccharomyces cerevisiae

TL;DR: Recent advances and new insights about MAPK-based signaling that have been made through studies in yeast are highlighted, which provide lessons directly applicable to, and that enhance the understanding of,MAPK-mediated signaling in mammalian cells.
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Genetic and pharmacological suppression of oncogenic mutations in ras genes of yeast and humans.

TL;DR: Observations establish a connection between the cholesterol biosynthetic pathway and transformation by the ras oncogene and offer a novel pharmacological approach to investigating, and possibly controlling, ras-mediated malignant transformations.