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Qi Zeng

Researcher at Agency for Science, Technology and Research

Publications -  73
Citations -  10404

Qi Zeng is an academic researcher from Agency for Science, Technology and Research. The author has contributed to research in topics: Metastasis & Cancer. The author has an hindex of 36, co-authored 71 publications receiving 9490 citations. Previous affiliations of Qi Zeng include National University of Singapore & Institute of Molecular and Cell Biology.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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A role for TAZ in migration, invasion, and tumorigenesis of breast cancer cells.

Siew Wee Chan, +7 more
- 15 Apr 2008 - 
TL;DR: The results indicate that TAZ plays a role in the migration, invasion, and tumorigenesis of breast cancer cells and thus presents a novel target for the detection and treatment of Breast cancer.
Journal ArticleDOI

The emerging role of the hippo pathway in cell contact inhibition, organ size control, and cancer development in mammals.

Qi Zeng, +1 more
- 11 Mar 2008 - 
TL;DR: The Hippo pathway defined originally in Drosophila melanogaster is conserved in mammals and is established as one major conserved mechanism governing cell contact inhibition, organ size control, and cancer development.
Journal Article

PRL-3 and PRL-1 promote cell migration, invasion, and metastasis.

Qi Zeng, +8 more
- 01 Jun 2003 - 
TL;DR: It is demonstrated that Chinese hamster ovary cells stably expressing PRL-3, a M(r) 20000 prenylated protein tyrosine phosphatase, or its relative,PRL-1, exhibit enhanced motility and invasive activity, and it is shown that PRl-3 is associated with diverse membrane structures involved in cell movement.
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PRL-3 down-regulates PTEN expression and signals through PI3K to promote epithelial-mesenchymal transition.

Haihe Wang, +5 more
- 01 Apr 2007 - 
TL;DR: Targeting PRL-3 may be a useful strategy to impede cancer cell invasion and metastasis because it could play an initiating role to trigger the EMT switch during cancer metastasis.