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Tatsuya Maeda

Researcher at University of Tokyo

Publications -  73
Citations -  12670

Tatsuya Maeda is an academic researcher from University of Tokyo. The author has contributed to research in topics: Saccharomyces cerevisiae & Kinase. The author has an hindex of 38, co-authored 69 publications receiving 11850 citations. Previous affiliations of Tatsuya Maeda include Hamamatsu University School of Medicine & Nagoya University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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A two-component system that regulates an osmosensing MAP kinase cascade in yeast

Tatsuya Maeda, +2 more
- 19 May 1994 - 
TL;DR: A two-component system in Saccharomyces cerevisiae that regulates an osmosensing MAP kinase cascade is described that contains an aspartate residue in the receiver domain of a cognate response regulator molecule.
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Yeast HOG1 MAP Kinase Cascade Is Regulated by a Multistep Phosphorelay Mechanism in the SLN1–YPD1–SSK1 “Two-Component” Osmosensor

Francesc Posas, +5 more
- 20 Sep 1996 - 
TL;DR: It is proposed that the multistep phosphorelay mechanism is a universal signal transduction apparatus utilized both in prokaryotes and eukaryotes.
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Activation of yeast PBS2 MAPKK by MAPKKKs or by binding of an SH3-containing osmosensor.

Tatsuya Maeda, +2 more
- 28 Jul 1995 - 
TL;DR: The role of mitogen-activated protein (MAP) kinase cascades in integrating distinct upstream signals was studied in yeast and mutants that were not able to activate PBS2 MAP kinase kinase (MAPKK; Pbs2p) at high osmolarity were characterized.
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Identification and characterization of Arabidopsis gibberellin receptors.

Masatoshi Nakajima, +12 more
- 01 Jun 2006 - 
TL;DR: All three AtGID1s functioned as GA receptors in Arabidopsis, and the expression of each AtG ID1 clone in the rice gid1-1 mutant rescued the GA-insensitive dwarf phenotype.