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Richard L. Proia

Researcher at National Institutes of Health

Publications -  222
Citations -  30484

Richard L. Proia is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Sphingosine & Receptor. The author has an hindex of 80, co-authored 212 publications receiving 28224 citations. Previous affiliations of Richard L. Proia include Merck & Co. & University of Texas Health Science Center at San Antonio.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Lymphocyte egress from thymus and peripheral lymphoid organs is dependent on S1P receptor 1

Mehrdad Matloubian, +8 more
- 22 Jan 2004 - 
TL;DR: It is established that S1P1 is essential for lymphocyte recirculation and that it regulates egress from both thymus and peripheral lymphoid organs.
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Edg-1, the G protein–coupled receptor for sphingosine-1-phosphate, is essential for vascular maturation

Yujing Liu, +13 more
- 15 Oct 2000 - 
TL;DR: The data reveal Edg-1 to be the first G protein-coupled receptor required for blood vessel formation and show that sphingolipid signaling is essential during mammalian development.
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Lysosomal Glycosphingolipid Recognition by NKT Cells

Dapeng Zhou, +16 more
- 03 Dec 2004 - 
TL;DR: It is suggested that expression of iGb3 in peripheral tissues may be involved in controlling NKT cell responses to infections and malignancy and in autoimmunity.
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Essential role for sphingosine kinases in neural and vascular development.

Kiyomi Mizugishi, +5 more
- 15 Dec 2005 - 
TL;DR: It is shown that S1P signaling is critical for neural and vascular development and joins a growing list of signaling molecules, such as vascular endothelial growth factor, which regulate the functionally intertwined pathways of angiogenesis and neurogenesis.