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Dmitry V. Bulavin

Researcher at French Institute of Health and Medical Research

Publications -  68
Citations -  11958

Dmitry V. Bulavin is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Phosphorylation & Kinase. The author has an hindex of 37, co-authored 66 publications receiving 11034 citations. Previous affiliations of Dmitry V. Bulavin include Institute of Molecular and Cell Biology & University of Nice Sophia Antipolis.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Phosphorylation of human p53 by p38 kinase coordinates N‐terminal phosphorylation and apoptosis in response to UV radiation

Dmitry V. Bulavin, +7 more
- 01 Dec 1999 - 
TL;DR: Results suggest that p38 kinase plays a prominent role in an integrated regulation of N‐terminal phosphorylation that regulates p53‐mediated apoptosis after UV radiation.
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Initiation of a G2/M checkpoint after ultraviolet radiation requires p38 kinase

Dmitry V. Bulavin, +7 more
- 03 May 2001 - 
TL;DR: It is reported that p38 kinase has a critical role in the initiation of a G2 delay after ultraviolet radiation, and regulation of Cdc25B phosphorylation by p38 is a critical event for initiating the G2/M checkpoint after ultraviolet Radiation.
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Genomic instability in Gadd45a- deficient mice

M C Hollander, +16 more
- 01 Oct 1999 - 
TL;DR: The results indicate that Gadd45a is one component of the p53 pathway that contributes to the maintenance of genomic stability and may contribute to the aneuploidy.
Journal ArticleDOI

Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity

Dmitry V. Bulavin, +13 more
- 20 May 2002 - 
TL;DR: Findings suggest that inactivation of the p38 MAPK through PPM1D overexpression resulting from P PM1D amplification contributes to the development of human cancers by suppressing p53 activation.