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Marc Lecuit

Researcher at Pasteur Institute

Publications -  346
Citations -  29687

Marc Lecuit is an academic researcher from Pasteur Institute. The author has contributed to research in topics: Listeria monocytogenes & Listeria. The author has an hindex of 83, co-authored 331 publications receiving 25601 citations. Previous affiliations of Marc Lecuit include Centre national de la recherche scientifique & Paris Descartes University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The Listeria transcriptional landscape from saprophytism to virulence

Alejandro Toledo-Arana, +32 more
- 18 Jun 2009 - 
TL;DR: Using tiling arrays and RNAs from wild-type and mutant bacteria grown in vitro, ex vivo and in vivo, the transcription of its entire genome is analysed and it is discovered that riboswitches can act as terminators for upstream genes.
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Chikungunya Virus and the Global Spread of a Mosquito-Borne Disease

Scott C. Weaver, +2 more
- 25 Mar 2015 - 
TL;DR: Chikungunya virus infection is a rapid-onset, febrile disease with intense asthenia, arthralgia, myalgia, headache, and rash.
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A transgenic model for listeriosis: role of internalin in crossing the intestinal barrier.

Marc Lecuit, +7 more
- 01 Jun 2001 - 
TL;DR: In guinea pigs and transgenic mice expressing human E-cadherin, internalin was found to mediate invasion of enterocytes and crossing of the intestinal barrier, illustrating how relevant animal models for human infections can be generated.
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A mouse model for Chikungunya: young age and inefficient type-I interferon signaling are risk factors for severe disease.

Thérèse Couderc, +14 more
- 08 Feb 2008 - 
TL;DR: It is shown that whereas wild type (WT) adult mice are resistant to CHIKV infection, WT mouse neonates are susceptible and neonatal disease severity is age-dependent, and the neonatal phase and inefficient type-I IFN signaling as risk factors for severe CHikV-associated disease are identified.