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Jin-Tai Yu

Researcher at Fudan University

Publications -  439
Citations -  26147

Jin-Tai Yu is an academic researcher from Fudan University. The author has contributed to research in topics: Single-nucleotide polymorphism & Dementia. The author has an hindex of 66, co-authored 439 publications receiving 20020 citations. Previous affiliations of Jin-Tai Yu include National Health and Family Planning Commission & Ocean University of China.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease.

TL;DR: The mechanisms and important role of pro-inflammatory cytokines in the pathogenesis of AD, and the ongoing drug targeting pro- inflammatory cytokine for therapeutic modulation are discussed.
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The prevalence of neuropsychiatric symptoms in Alzheimer's disease: Systematic review and meta-analysis

TL;DR: NPS were observed to be highly prevalent in AD patients, and disease duration, age, education level, population origin and the severity of cognitive impairment had influence on the prevalence of some NPS.
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Post-stroke cognitive impairment: epidemiology, mechanisms and management

TL;DR: The epidemiological features and the mechanisms of post-stroke cognitive impairment are described, the promising management strategies for these patients are discussed, and the treatments may benefit from the anti-dementia drugs, but also the manage measures on cerebrovascular diseases.
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Meta-analysis of modifiable risk factors for Alzheimer's disease

TL;DR: Effective interventions in diet, medications, biochemical exposures, psychological condition, pre-existing disease and lifestyle may decrease new incidence of AD.