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Hua Cheng

Researcher at University of Maryland, Baltimore

Publications -  17
Citations -  5592

Hua Cheng is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: IκB kinase & T cell. The author has an hindex of 10, co-authored 16 publications receiving 5124 citations. Previous affiliations of Hua Cheng include Penn State Cancer Institute & Pennsylvania State University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

HTLV-1 Tax Is a Critical Lipid Raft Modulator That Hijacks IκB Kinases to the Microdomains for Persistent Activation of NF-κB

Jiannan Huang, +6 more
- 06 Mar 2009 - 
TL;DR: Findings indicate that Tax1 actively recruits IKK to the lipid raft microdomains for persistent activation of NF-κB, thereby contributing to HTLV-1 oncogenesis.
Journal ArticleDOI

HTLV-1 Tax deregulates autophagy by recruiting autophagic molecules into lipid raft microdomains.

Tong Ren, +6 more
- 15 Jan 2015 - 
TL;DR: It is reported that Tax is a determining factor for dysregulation of autophagy in HTLV-1-transformed T cells and Tax-immortalized CD4 memory T cells, indicating a critical role of Tax-deregulated autophagosomes in promoting survival and transformation of virally infected T cells.