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Kailash Gulshan

Researcher at Cleveland Clinic

Publications -  36
Citations -  6487

Kailash Gulshan is an academic researcher from Cleveland Clinic. The author has contributed to research in topics: Inflammasome & ABCA1. The author has an hindex of 16, co-authored 29 publications receiving 5861 citations. Previous affiliations of Kailash Gulshan include Cleveland State University & University of Iowa.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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A nuclear receptor-like pathway regulating multidrug resistance in fungi

Jitendra K. Thakur, +14 more
- 03 Apr 2008 - 
TL;DR: Detailed mechanistic understanding of a fungal nuclear receptor-like gene regulatory pathway provides novel therapeutic targets for the treatment of multidrug-resistant fungal infections.
Journal ArticleDOI

Multidrug Resistance in Fungi

Kailash Gulshan, +1 more
- 01 Nov 2007 - 
TL;DR: One of the major advances serving to define the beginning of the era of modern medicine was the development of penicillin in the early 1940s as the first widely used antibiotic effective against microorganisms.
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Ceramide as a Mediator of Non-Alcoholic Fatty Liver Disease and Associated Atherosclerosis

Takhar Kasumov, +9 more
- 20 May 2015 - 
TL;DR: Modulation of ceramide synthesis may lead to the development of novel strategies for the treatment of both NAFLD and its associated atherosclerosis.
Journal ArticleDOI

Oxidant-specific Folding of Yap1p Regulates Both Transcriptional Activation and Nuclear Localization *

Kailash Gulshan, +3 more
- 09 Dec 2005 - 
TL;DR: It is demonstrated that the proper folding of Yap1p in the presence of H2O2 is required for recruitment of the mediator component Rox3p to the TRX2 promoter in addition to the nuclear accumulation of YAP1p during stress by this oxidant.