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Consiglia Pacelli

Researcher at University of Foggia

Publications -  47
Citations -  7028

Consiglia Pacelli is an academic researcher from University of Foggia. The author has contributed to research in topics: Mitochondrion & Oxidative phosphorylation. The author has an hindex of 20, co-authored 42 publications receiving 6172 citations. Previous affiliations of Consiglia Pacelli include Université de Montréal & University of Bari.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Elevated Mitochondrial Bioenergetics and Axonal Arborization Size Are Key Contributors to the Vulnerability of Dopamine Neurons.

Consiglia Pacelli, +5 more
- 21 Sep 2015 - 
TL;DR: This work shows for the first time that vulnerable nigral DA neurons differ from less vulnerable DA neurons by having a higher basal rate of mitochondrial OXPHOS, a smaller reserve capacity, a higher density of axonal mitochondria, an elevated level of basal oxidative stress, and a considerably more complex axonal arborization.
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Effect of resveratrol on mitochondrial function: implications in parkin-associated familiar Parkinson's disease

Anna Ferretta, +13 more
- 01 Jul 2014 - 
TL;DR: The results, obtained in early-onset PD fibroblasts, suggest that resveratrol may have potential clinical application in selected cases of PD-affected patients and alter PGC-1α activity and transcriptional deregulation of its target genes in PD pathogenesis.
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Effects of Serine 129 Phosphorylation on α-Synuclein Aggregation, Membrane Association, and Internalization

Filsy Samuel, +8 more
- 26 Feb 2016 - 
TL;DR: The results suggest that the threshold for vesicle permeabilization is evident even at low levels of α-syn internalization and are relevant to therapeutic strategies to reduce intercellular propagation ofα-syn misfolding.
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Mitochondrial defect and PGC-1α dysfunction in parkin-associated familial Parkinson's disease

Consiglia Pacelli, +15 more
- 01 Aug 2011 - 
TL;DR: The data confirm the role of parkin in mitochondrial homeostasis and suggest a potential involvement of the PGC-1α pathway in the pathogenesis of Parkinson's disease.