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Flaminia Pavone

Researcher at National Research Council

Publications -  122
Citations -  8817

Flaminia Pavone is an academic researcher from National Research Council. The author has contributed to research in topics: Neuropathic pain & Nociception. The author has an hindex of 35, co-authored 120 publications receiving 8044 citations. Previous affiliations of Flaminia Pavone include Novartis & Nagasaki University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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'Tissue' transglutaminase ablation reduces neuronal death and prolongs survival in a mouse model of Huntington's disease.

Pier G. Mastroberardino, +10 more
- 31 Aug 2002 - 
TL;DR: By crossing Huntington's disease R6/1 transgenic mice with ‘tissue’ transglutaminase (TG2) knock-out mice, it is demonstrated that this multifunctional enzyme plays an important role in the neuronal death characterising this disorder in vivo.
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TLQP-21, a VGF-derived peptide, increases energy expenditure and prevents the early phase of diet-induced obesity

Alessandro Bartolomucci, +24 more
- 26 Sep 2006 - 
TL;DR: Biochemical and molecular analyses suggest that TLQP-21 exerts its effects by stimulating autonomic activation of adrenal medulla and adipose tissues and prove that its chronic i.c.v. infusion effected an increase in EE and limited the early phase of diet-induced obesity.
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The function neutralizing anti-TrkA antibody MNAC13 reduces inflammatory and neuropathic pain.

Gabriele Ugolini, +4 more
- 20 Feb 2007 - 
TL;DR: It is demonstrated that MNAC13, the only anti-TrkA monoclonal antibody for which function neutralizing properties have been clearly shown both in vitro and in vivo, induces analgesia in both inflammatory and neuropathic pain models, with a surprisingly long-lasting effect in the latter.
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The analgesic effect on neuropathic pain of retrogradely transported botulinum neurotoxin A involves Schwann cells and astrocytes.

Sara Marinelli, +6 more
- 24 Oct 2012 - 
TL;DR: It is found that BoNT/A modulates the proliferation of SC and inhibits the acetylcholine release from SC, evidencing a new biological effect of the toxin and further supporting the retrograde transport of the toxins along the nerve and its ability to influence regenerative processes.