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François Bertrand Favier

Researcher at University of Montpellier

Publications -  25
Citations -  6113

François Bertrand Favier is an academic researcher from University of Montpellier. The author has contributed to research in topics: Skeletal muscle & Hypoxia (medical). The author has an hindex of 15, co-authored 22 publications receiving 5532 citations. Previous affiliations of François Bertrand Favier include Institut national de la recherche agronomique & Jean Monnet University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Cellular and molecular events controlling skeletal muscle mass in response to altered use.

François Bertrand Favier, +2 more
- 12 Jan 2008 - 
TL;DR: This review examines the current understanding of the cellular and molecular events involved in the control of muscle mass under conditions of muscle use and disuse, with particular attention to the effects of resistance exercise/training.
Journal ArticleDOI

Downregulation of Akt/mammalian target of rapamycin pathway in skeletal muscle is associated with increased REDD1 expression in response to chronic hypoxia.

François Bertrand Favier, +8 more
- 01 Jun 2010 - 
TL;DR: RedD1 is identified as a negative regulator of skeletal muscle mass during chronic hypoxia and Translation of this fundamental knowledge into the clinical investigation of COPD shows the interest to develop therapeutic strategies aimed at inhibiting REDD1.
Journal ArticleDOI

HIF-1-driven skeletal muscle adaptations to chronic hypoxia: molecular insights into muscle physiology

François Bertrand Favier, +7 more
- 23 Aug 2015 - 
TL;DR: Results demonstrate that skeletal muscle copes with the energetic challenge imposed by O2 rarefaction via metabolic optimization through adaptations to hypoxia that fit with HIF-1 activation.
Journal ArticleDOI

REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy

Florian Britto, +9 more
- 01 Dec 2014 - 
TL;DR: In vivo results demonstrated in vivo that REDD1 is required for glucocorticoid-induced inhibition of protein synthesis via mTORC1 downregulation, and highlighted a new mechanism involved in m TORC1 inhibition linking REDD 1, Akt, and PRAS40.